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TIQ-A
CAS No. 420849-22-5
TIQ-A( —— )
Catalog No. M28456 CAS No. 420849-22-5
TIQ-A is a PARP1 inhibitor which involved in DNA single-strand break repair via the base excision repair pathway. PARP1 is triggered by DNA damage and its excessive activation has been proposed as a causative factor in many pathological conditions including ischemia and reperfusion injury, asthma-related inflammation, and atherogenesis.
Purity : >98% (HPLC)
COA
Datasheet
HNMR
HPLC
MSDS
Handing Instructions
| Size | Price / USD | Stock | Quantity |
| 5MG | 147 | Get Quote |
|
| 10MG | 260 | Get Quote |
|
| 25MG | 484 | Get Quote |
|
| 50MG | 691 | Get Quote |
|
| 100MG | 888 | Get Quote |
|
| 500MG | 1872 | Get Quote |
|
| 1G | Get Quote | Get Quote |
|
Biological Information
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Product NameTIQ-A
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NoteResearch use only, not for human use.
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Brief DescriptionTIQ-A is a PARP1 inhibitor which involved in DNA single-strand break repair via the base excision repair pathway. PARP1 is triggered by DNA damage and its excessive activation has been proposed as a causative factor in many pathological conditions including ischemia and reperfusion injury, asthma-related inflammation, and atherogenesis.
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DescriptionTIQ-A is a PARP1 inhibitor which involved in DNA single-strand break repair via the base excision repair pathway. PARP1 is triggered by DNA damage and its excessive activation has been proposed as a causative factor in many pathological conditions including ischemia and reperfusion injury, asthma-related inflammation, and atherogenesis.
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In Vitro——
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In Vivo——
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Synonyms——
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PathwayCell Cycle/DNA Damage
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TargetPARP
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RecptorErα|NF-κB
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Research Area——
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Indication——
Chemical Information
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CAS Number420849-22-5
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Formula Weight201.24
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Molecular FormulaC11H7NOS
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Purity>98% (HPLC)
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SolubilityIn Vitro:?DMSO : 125 mg/mL (621.15 mM)
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SMILESO=c1[nH]c2sccc2c2ccccc12
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Chemical Name——
Shipping & Storage Information
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Storage(-20℃)
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ShippingWith Ice Pack
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Stability≥ 2 years
Reference
molnova catalog
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TIQ-A
TIQ-A is a PARP1 inhibitor which involved in DNA single-strand break repair via the base excision repair pathway. PARP1 is triggered by DNA damage and its excessive activation has been proposed as a causative factor in many pathological conditions including ischemia and reperfusion injury, asthma-related inflammation, and atherogenesis.
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