Methyl 3-phenylpropanoate

CAS No. 103-25-3

Methyl 3-phenylpropanoate( 3-Phenylpropionic acid methyl ester )

Catalog No. M28729 CAS No. 103-25-3

Methyl 3-phenylpropanoate is an endogenous metabolite.

Purity : >98% (HPLC)

COA Datasheet HNMR HPLC MSDS Handing Instructions
Size Price / USD Stock Quantity
1 mL x 10 mM in DMSO 28 In Stock
100MG Get Quote In Stock
200MG Get Quote In Stock
500MG Get Quote In Stock
1G 27 In Stock

Biological Information

  • Product Name
    Methyl 3-phenylpropanoate
  • Note
    Research use only, not for human use.
  • Brief Description
    Methyl 3-phenylpropanoate is an endogenous metabolite.
  • Description
    Methyl 3-phenylpropanoate is an endogenous metabolite.
  • In Vitro
    ——
  • In Vivo
    ——
  • Synonyms
    3-Phenylpropionic acid methyl ester
  • Pathway
    Proteasome/Ubiquitin
  • Target
    Endogenous Metabolite
  • Recptor
    ——
  • Research Area
    ——
  • Indication
    ——

Chemical Information

  • CAS Number
    103-25-3
  • Formula Weight
    164.204
  • Molecular Formula
    C10H12O2
  • Purity
    >98% (HPLC)
  • Solubility
    In Vitro:?DMSO : 100 mg/mL (609.01 mM)
  • SMILES
    COC(=O)CCc1ccccc1
  • Chemical Name
    ——

Shipping & Storage Information

  • Storage
    (-20℃)
  • Shipping
    With Ice Pack
  • Stability
    ≥ 2 years

Reference

1.Cao M , et al. Novel synthetic pyridyl analogues of CDDO-Imidazolide are useful new tools in cancer prevention. Pharmacol Res. 2015 Oct;100:135-47.
molnova catalog
related products
  • Neopterin

    D-(+)-Neopterin is produced by human monocytes/macrophages upon stimulation with the cytokine interferon-γ. In humans, neopterin is involved in purine biosynthesis.

  • 3-(3,4,5-Trimethoxyp...

    3-(3,4,5-Trimethoxyphenyl)-propanoic acid is a constituent of Piper retrofractum and Piper longum and is found in herbs and spices.

  • Butyric acid

    Butyric acid is a fatty acid isolated from many dairy products that exhibits anticancer activity. Butyric acid inhibits histone deacetylases (HDACs) and induces apoptosis and G1 phase cell cycle arrest in glioma cells.