PK095

CAS No. 380314-37-4

PK095( —— )

Catalog No. M35967 CAS No. 380314-37-4

PK095 is a proprietary compound in the guanidine - based F1F0-ATPase inhibitor family.

Purity : >98% (HPLC)

COA Datasheet HNMR HPLC MSDS Handing Instructions
Size Price / USD Stock Quantity
1 mL x 10 mM in DMSO 141 In Stock
5MG 139 In Stock
10MG 197 In Stock
25MG 314 In Stock
50MG 425 In Stock
100MG 574 In Stock
200MG 768 In Stock
500MG Get Quote In Stock
1G Get Quote In Stock

Biological Information

  • Product Name
    PK095
  • Note
    Research use only, not for human use.
  • Brief Description
    PK095 is a proprietary compound in the guanidine - based F1F0-ATPase inhibitor family.
  • Description
    PK095 is a p53 mutant stabilizer. PK095 can be used for research of cancer.
  • In Vitro
    ——
  • In Vivo
    ——
  • Synonyms
    ——
  • Pathway
    Endocrinology/Hormones
  • Target
    ATPase
  • Recptor
    ATPase
  • Research Area
    ——
  • Indication
    ——

Chemical Information

  • CAS Number
    380314-37-4
  • Formula Weight
    378.45
  • Molecular Formula
    C20H18N4O2S
  • Purity
    >98% (HPLC)
  • Solubility
    ——
  • SMILES
    C(C)N1C=2C(C=3C1=CC=CC3)=CC(NC(CSC=4NC(=O)C=CN4)=O)=CC2
  • Chemical Name
    ——

Shipping & Storage Information

  • Storage
    (-20℃)
  • Shipping
    With Ice Pack
  • Stability
    ≥ 2 years

Reference

1. Alan Fersht, et al. Compounds for use in stabilizing p53 mutants. Patent. WO2009136175.
molnova catalog
related products
  • Esomeprazole Magnesi...

    Esomeprazole Magnesium is a proton pump inhibitor to reduce gastric acid secretion.

  • Reboxetine

    Reboxetine is an antidepressant drug used in the treatment of clinical depression, panic disorder and ADD/ADHD.

  • Mtb ATP synthase-IN-...

    Mtb ATP synthase-IN-1 (compound 6ab) is a potent inhibitor of Mycobacterium tuberculosis (Mtb) ATP synthase (MIC = 0.452-0.499 μg/mL) that can be used in anti-mycobacterium research, showing low cytotoxicity (Vero IC 50 > 64 μg/mL) and acceptable oral bioavailability with good metabolic stability.