α-Glucosidase-IN-4

CAS No. 2410538-67-7

α-Glucosidase-IN-4( —— )

Catalog No. M35462 CAS No. 2410538-67-7

α-Glucosidase-IN-4 is a potent α-glucosidase inhibitor exhibiting antihypertensive and hypoglycemic activity.

Purity : >98% (HPLC)

COA Datasheet HNMR HPLC MSDS Handing Instructions
Size Price / USD Stock Quantity
5MG 645 In Stock
10MG 798 In Stock
25MG 1055 In Stock
50MG 1283 In Stock
100MG 1628 In Stock
200MG 2195 In Stock
500MG Get Quote In Stock
1G Get Quote In Stock

Biological Information

  • Product Name
    α-Glucosidase-IN-4
  • Note
    Research use only, not for human use.
  • Brief Description
    α-Glucosidase-IN-4 is a potent α-glucosidase inhibitor exhibiting antihypertensive and hypoglycemic activity.
  • Description
    α-Glucosidase-IN-4 is a reversible and mixed type α-glucosidase inhibitor with an IC50 of 12.98 μM, a KI of 27.02 μM, and a KIS of 13.65 μM, respectively.
  • In Vitro
    ——
  • In Vivo
    ——
  • Synonyms
    ——
  • Pathway
    Others
  • Target
    Other Targets
  • Recptor
    Glucosidase
  • Research Area
    ——
  • Indication
    ——

Chemical Information

  • CAS Number
    2410538-67-7
  • Formula Weight
    306.31
  • Molecular Formula
    C19H14O4
  • Purity
    >98% (HPLC)
  • Solubility
    ——
  • SMILES
    O(C(/C=C/C1=CC=C(C)C=C1)=O)C=2C=3C(OC(=O)C2)=CC=CC3
  • Chemical Name
    ——

Shipping & Storage Information

  • Storage
    (-20℃)
  • Shipping
    With Ice Pack
  • Stability
    ≥ 2 years

Reference

1. Xu XT, et al. Synthesis and biological evaluation of coumarin derivatives as α-glucosidase inhibitors. Eur J Med Chem. 2020;189:112013.?
molnova catalog
related products
  • CTP Synthetase-IN-1

    CTP Synthetase-IN-1 is an orally active and potent cytidine 5'-triphosphate synthetase (CTPS) inhibitor with potential antitumour activity, shows anti-inflammatory activity in animal models of inflammation, inhibits human CTPS1 and human CTPS2, and can be used in studies of arthritis and rheumatoid arthritis.

  • AMDE-1

    AMDE-1 is a modulator of autophagy activity that acts by triggering autophagy in an Atg5-dependent manner, recruiting Atg16 to the pre-autophagosomal site and causing LC3 lipidation.

  • dTAG-47

    dTAG-47, heterobifunctional dTAG molecule, targets mutant FKBP12 (FKBP12F36V). FKBP12F36V serves as a degradation tag (dTAG) and is fused to a protein of interest. dTAG-47 can be used for the research of basal-like breast cancers (BBC).