Leukotriene E4
CAS No. 75715-89-8
Leukotriene E4( —— )
Catalog No. M35237 CAS No. 75715-89-8
Leukotriene E4 (LTE4) (LTE4) is produced by the action of dipeptidyl peptidase on LTD4 and is a component of the Slow Reactive Substance of Anaphylaxis (SRS-A).
Purity : >98% (HPLC)
COA
Datasheet
HNMR
HPLC
MSDS
Handing Instructions
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Biological Information
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Product NameLeukotriene E4
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NoteResearch use only, not for human use.
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Brief DescriptionLeukotriene E4 (LTE4) (LTE4) is produced by the action of dipeptidyl peptidase on LTD4 and is a component of the Slow Reactive Substance of Anaphylaxis (SRS-A).
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DescriptionLeukotriene E4 (LTE4) is produced by the action of dipeptidase on LTD4. Leukotriene E4 is one of the constituents of slow-reacting substance of anaphylaxis (SRS-A). Leukotriene E4 accumulates in both plasma and urine and urinary excretion of Leukotriene E4 is most often used as an indicator of asthma.
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In Vitro——
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In Vivo——
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Synonyms——
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PathwayProteasome/Ubiquitin
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TargetEndogenous Metabolite
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RecptorEndogenous Metabolite
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Research Area——
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Indication——
Chemical Information
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CAS Number75715-89-8
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Formula Weight439.61
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Molecular FormulaC23H37NO5S
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Purity>98% (HPLC)
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Solubility——
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SMILES[C@@H]([C@H](CCCC(O)=O)O)(SC[C@@H](C(O)=O)N)/C=C/C=C/C=C\C/C=C\CCCCC
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Chemical Name——
Shipping & Storage Information
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Storage(-20℃)
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ShippingWith Ice Pack
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Stability≥ 2 years
Reference
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INOSINIC ACID
Inosinic acid is a purine nucleotide which has hypoxanthine as the base and one phosphate group esterified to the sugar moiety.
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DL-Homocysteinethiol...
DL-Homocysteinethiolactone hydrochloride is a cysteine derivative that binds to and induces conformational changes in various plasma proteins slowing coagulation and inducing oxidative stress.?It decreases left ventricular systolic blood pressure and cardiac force and induces seizures in vivo.
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Lipoxin A4
Lipoxin A4 (LXA4) is a lipoxygenase-derived arachidonate-like mediator with anti-inflammatory properties.Lipoxin A4 attenuates MSU crystal-induced NLRP3 inflammasome activation through inhibition of Nrf2, and regulates M1/M2 macrophage polarization through the FPR2-IRF pathway.
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