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GIP (3-42), human
CAS No. 1802086-25-4
GIP (3-42), human( —— )
Catalog No. M34964 CAS No. 1802086-25-4
GIP (3-42), human (Gastric Inhibitory Polypeptide (3-42) (human)) is a peptide that acts as a glucose-dependent proinsulinotropic polypeptide (GIP) receptor antagonist and regulates insulin secretion and the metabolic effects of GIP in vivo, which can be used to study type 2 diabetes.
Purity : >98% (HPLC)
COA
Datasheet
HNMR
HPLC
MSDS
Handing Instructions
| Size | Price / USD | Stock | Quantity |
| 2MG | 298 | Get Quote |
|
| 5MG | 463 | Get Quote |
|
| 10MG | 661 | Get Quote |
|
| 25MG | 1003 | Get Quote |
|
| 50MG | 1371 | Get Quote |
|
| 100MG | 1782 | Get Quote |
|
| 500MG | Get Quote | Get Quote |
|
| 1G | Get Quote | Get Quote |
|
Biological Information
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Product NameGIP (3-42), human
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NoteResearch use only, not for human use.
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Brief DescriptionGIP (3-42), human (Gastric Inhibitory Polypeptide (3-42) (human)) is a peptide that acts as a glucose-dependent proinsulinotropic polypeptide (GIP) receptor antagonist and regulates insulin secretion and the metabolic effects of GIP in vivo, which can be used to study type 2 diabetes.
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DescriptionGIP (3-42), human acts as a glucose-dependent insulinotropic polypeptide (GIP) receptor antagonist, moderating the insulin secreting and metabolic actions of GIP in vivo.
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In VitroThe incretin hormone GIP is rapidly degraded in the circulation by dipeptidyl peptidase IV (DPP IV) forming the N-terminally truncated peptide GIP(3-42).
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In Vivo——
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Synonyms——
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PathwayAngiogenesis
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TargetIGF-1R
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RecptorIGF-1R
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Research Area——
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Indication——
Chemical Information
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CAS Number1802086-25-4
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Formula Weight4749.35
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Molecular FormulaC214H324N58O63S
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Purity>98% (HPLC)
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SolubilityIn Vitro:?DMSO : 100 mg/mL (21.06 mM; Ultrasonic )H2O : 50 mg/mL (10.53 mM; Ultrasonic)
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SMILES——
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Chemical Name——
Shipping & Storage Information
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Storage(-20℃)
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ShippingWith Ice Pack
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Stability≥ 2 years
Reference
1. V A Gault, et al. Evidence that the major degradation product of glucose-dependent insulinotropic polypeptide, GIP(3-42), is a GIP receptor antagonist in vivo. J Endocrinol. 2002 Nov;175(2):525-33.?
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