N-Oleoyl-L-Serine

CAS No. 107743-37-3

N-Oleoyl-L-Serine( —— )

Catalog No. M34817 CAS No. 107743-37-3

N-Oleoyl-L-Serine (N-Oleoylserine) is an endogenous long-chain fatty acid amide that is a lipid modulator of bone remodeling and stimulates osteoclast apoptosis which can be used to study osteoporosis.

Purity : >98% (HPLC)

COA Datasheet HNMR HPLC MSDS Handing Instructions
Size Price / USD Stock Quantity
5MG 185 In Stock
10MG 328 In Stock
25MG 554 In Stock
50MG 775 In Stock
100MG 1070 In Stock
200MG 1442 In Stock
500MG 2130 In Stock
1G Get Quote In Stock

Biological Information

  • Product Name
    N-Oleoyl-L-Serine
  • Note
    Research use only, not for human use.
  • Brief Description
    N-Oleoyl-L-Serine (N-Oleoylserine) is an endogenous long-chain fatty acid amide that is a lipid modulator of bone remodeling and stimulates osteoclast apoptosis which can be used to study osteoporosis.
  • Description
    N-Oleoyl-L-Serine is an endogenous amide of long-chain fatty acids with ethanolamine (N-acyl amides). N-Oleoyl-L-Serine is a lipid regulator of bone remodeling and stimulates osteoclast apoptosis. N-Oleoyl-L-Serine can be used for antiosteoporotic drug discovery development.
  • In Vitro
    ——
  • In Vivo
    ——
  • Synonyms
    ——
  • Pathway
    Apoptosis
  • Target
    Apoptosis
  • Recptor
    Apoptosis
  • Research Area
    ——
  • Indication
    ——

Chemical Information

  • CAS Number
    107743-37-3
  • Formula Weight
    369.54
  • Molecular Formula
    C21H39NO4
  • Purity
    >98% (HPLC)
  • Solubility
    ——
  • SMILES
    N([C@H](C(O)=O)CO)C(CCCCCCC/C=C\CCCCCCCC)=O
  • Chemical Name
    ——

Shipping & Storage Information

  • Storage
    (-20℃)
  • Shipping
    With Ice Pack
  • Stability
    ≥ 2 years

Reference

1. Reem Smoum, et al. Oleoyl serine, an endogenous N-acyl amide, modulates bone remodeling and mass. Proc Natl Acad Sci U S A. 2010 Oct 12;107(41):17710-5.?
molnova catalog
related products
  • TMI-1

    TMI-1 (WAY-171318), a novel orally active inhibitor of ADAM17 (TACE) and MMP, induces tumor apoptosis in a breast cancer.

  • AAPK-25

    AAPK-25, a potent and selective dual inhibitor of Aurora/PLK, causes mitotic delay and cell arrest in prometaphase, via phosphorylation of the biomarker histone H3Ser10, followed by a surge in apoptosis.

  • Neoisoliquiritin b

    Neoisoliquiritigenin, isolated from Spatholobus suberectus, inhibits cell proliferation and induces cell apoptosis in breast cancer by directly binding to GRP78 to regulate the β-catenin pathway