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Vazegepant HCl
CAS No. 1414976-20-7
Vazegepant HCl( —— )
Catalog No. M22411 CAS No. 1414976-20-7
Vazegepant is the first intranasal gepant for the acute treatment of migraine and has announced a positive phase II/III study.
Purity : >98% (HPLC)
COA
Datasheet
HNMR
HPLC
MSDS
Handing Instructions
| Size | Price / USD | Stock | Quantity |
| 1 mL x 10 mM in DMSO | 330 | In Stock |
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| 5MG | 227 | In Stock |
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| 10MG | 334 | In Stock |
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| 25MG | 565 | In Stock |
|
| 50MG | 789 | In Stock |
|
| 100MG | 1088 | In Stock |
|
| 200MG | Get Quote | In Stock |
|
| 500MG | Get Quote | In Stock |
|
| 1G | Get Quote | In Stock |
|
Biological Information
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Product NameVazegepant HCl
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NoteResearch use only, not for human use.
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Brief DescriptionVazegepant is the first intranasal gepant for the acute treatment of migraine and has announced a positive phase II/III study.
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DescriptionVazegepant is the first intranasal gepant for the acute treatment of migraine and has announced a positive phase II/III study.
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In Vitro——
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In Vivo——
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Synonyms——
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PathwayOthers
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TargetOther Targets
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RecptorOthers
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Research Area——
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Indication——
Chemical Information
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CAS Number1414976-20-7
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Formula Weight675.3
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Molecular FormulaC36H47ClN8O3
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Purity>98% (HPLC)
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Solubility——
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SMILESCl.CN1CCC(CC1)N1CCN(CC1)C(=O)[C@@H](Cc1cc(C)c2[nH]ncc2c1)NC(=O)N1CCC(CC1)c1cc2ccccc2[nH]c1=O
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Chemical Name——
Shipping & Storage Information
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Storage(-20℃)
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ShippingWith Ice Pack
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Stability≥ 2 years
Reference
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Moth Cytochrome C (M...
Moth Cytochrome C (MCC) (88-103), derived from the carboxyl terminus of moth cytochrome c, induces positive selection of TCR transgenic thymocytes.This peptide is derived from the carboxyl terminus of moth cytochrome c. Thymic positive and negative selections govern the development of a self-MHC-reactive, yet self-tolerant, T cell repertoire.
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Adenosine-5'-diphosp...
Adenosine 5'-diphosphate sodium salt is an adenine nucleotide that is phosphorylated into ATP by ATPase. This phosphorylation is a key part of cellular homeostatis as it allows for energy storage and is involved in nucleic acid metabolism. ADP affects platelet activation through its interaction with ADP receptors P2Y1 P2Y12 and P2X1.
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