N-Acetylputrescine hydrochloride
CAS No. 18233-70-0
N-Acetylputrescine hydrochloride( —— )
Catalog No. M20805 CAS No. 18233-70-0
N-Acetylputrescine is a polyamine commonly occurring excreted in normal human urine.
Purity : >98% (HPLC)
COA
Datasheet
HNMR
HPLC
MSDS
Handing Instructions
| Size | Price / USD | Stock | Quantity |
| 25MG | 51 | In Stock |
|
| 100MG | 133 | In Stock |
|
| 200MG | Get Quote | In Stock |
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| 500MG | Get Quote | In Stock |
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| 1G | Get Quote | In Stock |
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Biological Information
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Product NameN-Acetylputrescine hydrochloride
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NoteResearch use only, not for human use.
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Brief DescriptionN-Acetylputrescine is a polyamine commonly occurring excreted in normal human urine.
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DescriptionN-Acetylputrescine is a polyamine commonly occurring excreted in normal human urine.
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In Vitro——
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In Vivo——
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Synonyms——
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PathwayProteasome/Ubiquitin
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TargetEndogenous Metabolite
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RecptorHuman Endogenous Metabolite
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Research Area——
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Indication——
Chemical Information
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CAS Number18233-70-0
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Formula Weight166.65
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Molecular FormulaC6H15ClN2O?
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Purity>98% (HPLC)
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SolubilityIn Vitro:?H2O : 200 mg/mL (1200.12 mM)
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SMILESCC(=O)NCCCCN.Cl
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Chemical Name——
Shipping & Storage Information
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Storage(-20℃)
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ShippingWith Ice Pack
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Stability≥ 2 years
Reference
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Glyceryl trimyristat...
Within the cell glyceryl trimyristate is primarily located in the membrane (predicted from logP) and adiposome. glyceryl trimyristate exists in all eukaryotes ranging from yeast to humans. In humans glyceryl trimyristate is involved in the metabolic disorder called de novo triacylglycerol biosynthesis glyceryl trimyristate pathway.
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3-Methylglutaric aci...
Methylglutaric acid is a leucine metabolite. A large amount of methylglutaric acid is identified in urine of patients with deficiency of 3-methylglutaconyl coenzyme A hydratase. Methylglutaric acid is also found to be associated with 3-hydroxy-3-methylglutaryl-CoA lyase deficiency another inborn error of metabolism.
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Cyclo(his-pro)
Cyclo(his-pro) is an endogenous cyclic dipeptide that exerts oxidative damage protection by selectively activating the transcription factor Nrf2 signalling pathway.?
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