Cart
sales@molnova.com
D-(+)-Turanose
CAS No. 547-25-1
D-(+)-Turanose( —— )
Catalog No. M20273 CAS No. 547-25-1
D(+)-Turanose is a naturally occuring compound. It is a reducing disaccharide.
Purity : >98% (HPLC)
COA
Datasheet
HNMR
HPLC
MSDS
Handing Instructions
| Size | Price / USD | Stock | Quantity |
| 100MG | Get Quote | In Stock |
|
| 200MG | 33 | In Stock |
|
| 500MG | Get Quote | In Stock |
|
| 1G | Get Quote | In Stock |
|
Biological Information
-
Product NameD-(+)-Turanose
-
NoteResearch use only, not for human use.
-
Brief DescriptionD(+)-Turanose is a naturally occuring compound. It is a reducing disaccharide.
-
DescriptionD(+)-Turanose is a naturally occuring compound. It is a reducing disaccharide.
-
In Vitro——
-
In Vivo——
-
Synonyms——
-
PathwayProteasome/Ubiquitin
-
TargetEndogenous Metabolite
-
RecptorHuman Endogenous Metabolite
-
Research Area——
-
Indication——
Chemical Information
-
CAS Number547-25-1
-
Formula Weight342.3
-
Molecular FormulaC12H22O11
-
Purity>98% (HPLC)
-
SolubilityH2O:130 mg/mL (379.78 mM)
-
SMILESOC[C@H]1O[C@H](O[C@H]2[C@H](O)[C@H](O)COC2(O)CO)[C@H](O)[C@@H](O)[C@@H]1O
-
Chemical Name——
Shipping & Storage Information
-
Storage(-20℃)
-
ShippingWith Ice Pack
-
Stability≥ 2 years
Reference
-
Glyceryl trimyristat...
Within the cell glyceryl trimyristate is primarily located in the membrane (predicted from logP) and adiposome. glyceryl trimyristate exists in all eukaryotes ranging from yeast to humans. In humans glyceryl trimyristate is involved in the metabolic disorder called de novo triacylglycerol biosynthesis glyceryl trimyristate pathway.
-
cis-4-Hydroxy-L-prol...
cis-4-Hydroxy-L-proline is an inhibitor of the synthesis and extracellular deposition of collagen. cis-4-Hydroxy-L-proline could inhibit fibroblast growth by preventing the deposition of triple-helical collagen on the cell layer.
-
Lipoxin A4
Lipoxin A4 (LXA4) is a lipoxygenase-derived arachidonate-like mediator with anti-inflammatory properties.Lipoxin A4 attenuates MSU crystal-induced NLRP3 inflammasome activation through inhibition of Nrf2, and regulates M1/M2 macrophage polarization through the FPR2-IRF pathway.
