α-Cyclodextrin
CAS No. 10016-20-3
α-Cyclodextrin( NSC 269470 | NSC-269470 | NSC269470 | Alfadex )
Catalog No. M26868 CAS No. 10016-20-3
Alfadex is a soluble fiber derived from corn and has beneficial effects on weight management in obese individuals with type 2 diabetes.
Purity : >98% (HPLC)
COA
Datasheet
HNMR
HPLC
MSDS
Handing Instructions
| Size | Price / USD | Stock | Quantity |
| 1G | 38 | In Stock |
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Biological Information
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Product Nameα-Cyclodextrin
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NoteResearch use only, not for human use.
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Brief DescriptionAlfadex is a soluble fiber derived from corn and has beneficial effects on weight management in obese individuals with type 2 diabetes.
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DescriptionAlfadex is a soluble fiber derived from corn and has beneficial effects on weight management in obese individuals with type 2 diabetes.(In Vivo):Alfadex preferentially reduces blood levels of saturated and trans fats in the LDL receptor knockout mice. Two independent in vivo biological test systems were used, a single cell Xenopus oocyte with expressed human aquaporin for cell hydration studies and the nematode Caenorhabditis elegans for testing life span in the treated animals. Alfadex was found to enhance water uptake through aquaporins of oocytes. Furthermore, Alfadex promoted longevity in C. elegans.
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In Vitro——
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In Vivo——
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SynonymsNSC 269470 | NSC-269470 | NSC269470 | Alfadex
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PathwayOthers
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TargetOther Targets
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RecptorSET-PP2A
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Research Area——
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Indication——
Chemical Information
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CAS Number10016-20-3
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Formula Weight972.846
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Molecular FormulaC36H60O30
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Purity>98% (HPLC)
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SolubilityIn Vitro:?DMSO : 110 mg/mL (113.07 mM)
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SMILESOC[C@H]1O[C@@H]2O[C@@H]3[C@@H](CO)O[C@H](O[C@@H]4[C@@H](CO)O[C@H](O[C@@H]5[C@@H](CO)O[C@H](O[C@@H]6[C@@H](CO)O[C@H](O[C@@H]7[C@@H](CO)O[C@H](O[C@H]1[C@H](O)[C@H]2O)[C@H](O)[C@H]7O)[C@H](O)[C@H]6O)[C@H](O)[C@H]5O)[C@H](O)[C@H]4O)[C@H](O)[C@H]3O
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Chemical Name——
Shipping & Storage Information
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Storage(-20℃)
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ShippingWith Ice Pack
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Stability≥ 2 years
Reference
1.Zonta F, et al. Lyn sustains oncogenic signaling in chronic lymphocytic leukemia by strengthening SET-mediated inhibition of PP2A. Blood. 2015;125(24):3747-3755.
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