Cart
sales@molnova.com
Dendroaspis Natriuretic Peptide
CAS No. 255721-52-9
Dendroaspis Natriuretic Peptide( ——— )
Catalog No. M39912 CAS No. 255721-52-9
Dendroaspis Natriuretic Peptide is a vasodilator peptide that can be isolated from animal venom.
Purity : >98% (HPLC)
COA
Datasheet
HNMR
HPLC
MSDS
Handing Instructions
| Size | Price / USD | Stock | Quantity |
| 25MG | Get Quote | Get Quote |
|
| 50MG | Get Quote | Get Quote |
|
| 100MG | Get Quote | Get Quote |
|
| 200MG | Get Quote | Get Quote |
|
| 500MG | Get Quote | Get Quote |
|
| 1G | Get Quote | Get Quote |
|
Biological Information
-
Product NameDendroaspis Natriuretic Peptide
-
NoteResearch use only, not for human use.
-
Brief DescriptionDendroaspis Natriuretic Peptide is a vasodilator peptide that can be isolated from animal venom.
-
DescriptionDendroaspis Natriuretic Peptide is a vasodilator peptide that can be isolated from animal venom.
-
In Vitro———
-
In Vivo———
-
Synonyms———
-
PathwayOthers
-
TargetOther Targets
-
Recptor———
-
Research Area———
-
Indication———
Chemical Information
-
CAS Number255721-52-9
-
Formula Weight4190.72
-
Molecular FormulaC180H282N56O56S2
-
Purity>98% (HPLC)
-
Solubility———
-
SMILES———
-
Chemical Name——
Shipping & Storage Information
-
Storage(-20℃)
-
ShippingWith Ice Pack
-
Stability≥ 2 years
Reference
1. Alves RS, et al. Isolation, homology modeling and renal effects of a C-type natriuretic peptide from the venom of the Brazilian yellow scorpion (Tityus serrulatus). Toxicon. 2013 Nov;74:19-26.?
molnova catalog
related products
-
EML4-ALK kinase inhi...
EML4-ALK kinase inhibitor 1 is a potent oral active inhibitor of echinoderm microtubule-associated protein-like 4-anaplastic lymphoma kinase (EML4-ALK), with an IC50 of 1 nM.
-
Phytone
Phytone enhanced both the sticky materials production and cell growth ?plays some role in the increase of sticky materials and cell growth.
-
Cyclic MKEY TFA (934...
Cyclic MKEY TFA is a synthetic cyclic peptide inhibitor of CXCL4-CCL5 heterodimer formation, which protects against atherosclerosis and aortic aneurysm formation by mediating inflammation.
