Home - Products - Neuroscience - GluR - gamma-DGG acetate(6729-55-1 free base)

gamma-DGG acetate(6729-55-1 free base)

CAS No. 2935387-13-4

gamma-DGG acetate(6729-55-1 free base)( —— )

Catalog No. M33365 CAS No. 2935387-13-4

gamma-DGG acetate(6729-55-1 free base) (γ-D-Glutamylglycine) is a competitive blocker of AMPA receptor.

Purity : >98% (HPLC)

COA Datasheet HNMR HPLC MSDS Handing Instructions
Size Price / USD Stock Quantity
25MG 37 In Stock
50MG 45 In Stock
100MG Get Quote In Stock
200MG Get Quote In Stock
500MG Get Quote In Stock
1G Get Quote In Stock

Biological Information

  • Product Name
    gamma-DGG acetate(6729-55-1 free base)
  • Note
    Research use only, not for human use.
  • Brief Description
    gamma-DGG acetate(6729-55-1 free base) (γ-D-Glutamylglycine) is a competitive blocker of AMPA receptor.
  • Description
    gamma-DGG acetate(6729-55-1 free base) (γ-D-Glutamylglycine) is a competitive blocker of AMPA receptor.
  • In Vitro
    ——
  • In Vivo
    ——
  • Synonyms
    ——
  • Pathway
    Neuroscience
  • Target
    GluR
  • Recptor
    GluR
  • Research Area
    ——
  • Indication
    ——

Chemical Information

  • CAS Number
    2935387-13-4
  • Formula Weight
    264.23
  • Molecular Formula
    C9H16N2O7
  • Purity
    >98% (HPLC)
  • Solubility
    ——
  • SMILES
    CC(O)=O.O=C(O)CNC(CC[C@H](C(O)=O)N)=O
  • Chemical Name
    ——

Shipping & Storage Information

  • Storage
    (-20℃)
  • Shipping
    With Ice Pack
  • Stability
    ≥ 2 years

Reference

molnova catalog
related products
  • FPTQ B

    FPTQ is a highly effective antagonist of mGluR 1, displaying IC50 values of 6 nM and 1.4 nM for human and mouse mGluR1 respectively . In addition to its robust inhibitory properties, FPTQ exhibits notable antioxidant and anti-inflammatory effects in both in vitro and in vivo settings .

  • IDRA-21

    IDRA-21 is a positive AMPA receptor modulator.?

  • NS-102

    NS-102 is a glutamate receptor and NMDA receptor antagonist that inhibits erythrocyanine (GluK2), which reduces glur6 receptor-mediated currents, and inhibits specific binding to the glur6 receptor.