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Calmodulin-Dependent Protein Kinase II 281-309
CAS No. 116826-37-0
Calmodulin-Dependent Protein Kinase II 281-309( —— )
Catalog No. M30414 CAS No. 116826-37-0
Calmodulin-Dependent Protein Kinase II (281-309) is a synthetic peptide that can be phosphorylated at Thr286 by PKC and inhibits CaM kinase II (IC50 = 80 nM).
Purity : >98% (HPLC)
COA
Datasheet
HNMR
HPLC
MSDS
Handing Instructions
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Biological Information
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Product NameCalmodulin-Dependent Protein Kinase II 281-309
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NoteResearch use only, not for human use.
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Brief DescriptionCalmodulin-Dependent Protein Kinase II (281-309) is a synthetic peptide that can be phosphorylated at Thr286 by PKC and inhibits CaM kinase II (IC50 = 80 nM).
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DescriptionCalmodulin-Dependent Protein Kinase II (281-309) is a synthetic peptide that can be phosphorylated at Thr286 by PKC and inhibits CaM kinase II (IC50 = 80 nM).
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In Vitro——
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In Vivo——
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Synonyms——
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PathwayOthers
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TargetOther Targets
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Recptor——
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Research Area——
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Indication——
Chemical Information
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CAS Number116826-37-0
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Formula Weight3374.06
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Molecular FormulaC146H254N46O39S3
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Purity>98% (HPLC)
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Solubility——
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SMILES——
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Chemical NameSequence:{Met}{His}{Arg}{Gln}{Glu}{Thr}{Val}{Asp}{Cys}{Leu}{Lys}{Lys}{Phe}{Asn}{Ala}{Arg}{Arg}{Lys}{Leu}{Lys}{Gly}{Ala}{Ile}{Leu}{Thr}{Thr}{Met}{Leu}{Ala}
Shipping & Storage Information
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Storage(-20℃)
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ShippingWith Ice Pack
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Stability≥ 2 years
Reference
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Kauniolide
Kauniolide(81066-45-7) is a natural compound.
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Suriclone
Suriclone (RP 31264) is a selective cyclic pyrrolidone analog with sedative and anxiolytic activity, but not significant sedative effects.Suriclone shows its effects by modulating GABA-A receptors.
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Nogo-66 (1-40)
Peptide fragment corresponding to residues 1 - 40 of Nogo-66, the domain of the myelin protein Nogo that inhibits axonal outgrowth. Acts as a competitive antagonist at the Nogo-66 receptor (NgR); blocks Nogo-66- and CNS myelin-induced inhibition of axonal growth, but does not reduce myelin-associated glycoprotein (MAG) inhibition of neurite outgrowth in vitro. Promotes regeneration of hemisected spinal axons and locomotor recovery following spinal injury in vivo.
