STK414603
CAS No. 796115-75-8
STK414603( —— )
Catalog No. M28974 CAS No. 796115-75-8
STK414603 is a heterocyclic acetamide derivative with potential analgesic, anti-inflammatory, and antimicrobial activities.
Purity : >98% (HPLC)
COA
Datasheet
HNMR
HPLC
MSDS
Handing Instructions
| Size | Price / USD | Stock | Quantity |
| 1 mL x 10 mM in DMSO | 29 | In Stock |
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| 10MG | 34 | In Stock |
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| 25MG | 54 | In Stock |
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| 50MG | 77 | In Stock |
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| 100MG | 113 | In Stock |
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| 200MG | Get Quote | In Stock |
|
| 500MG | Get Quote | In Stock |
|
| 1G | Get Quote | In Stock |
|
Biological Information
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Product NameSTK414603
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NoteResearch use only, not for human use.
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Brief DescriptionSTK414603 is a heterocyclic acetamide derivative with potential analgesic, anti-inflammatory, and antimicrobial activities.
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DescriptionSTK414603 is a heterocyclic acetamide derivative with potential analgesic, anti-inflammatory, and antimicrobial activities.
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In Vitro——
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In Vivo——
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Synonyms——
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PathwayOthers
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TargetOther Targets
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RecptorAromatase|CYP19
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Research Area——
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Indication——
Chemical Information
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CAS Number796115-75-8
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Formula Weight252.72
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Molecular FormulaC11H9ClN2OS
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Purity>98% (HPLC)
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Solubility——
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SMILESO=C(Cc(cccc1)c1Cl)Nc1nccs1
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Chemical Name——
Shipping & Storage Information
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Storage(-20℃)
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ShippingWith Ice Pack
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Stability≥ 2 years
Reference
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Proanthocyanidins
Extracted from Grape seed,pine bark coast of France; Suitability:Water,most organic solvents;Store the product in sealed,cool and dry condition.
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Araloside X
Aralosides possess protective effect against experimental myocardial ischemia and infarction, the mechanism of myocardial protection may be attributed to the amelioration of FFA metabolic deterioration and membrane peroxidation induced by oxygen free radicals. Aralosides have anti-inflammatory effect, they also can effectively prevent acute alcoholic liver injury.
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PTD-p65-P1 Peptide
PTD-p65-P1 Peptide is a nuclear transcription factor NF-kappaB inhibitor, composed of a membrane-translocating peptide sequence generated from antennapedia (PTD) conjugated with p65-P1, which selectively inhibits activation induced by various inflammatory stimuli. PTD-p65-P1 suppressed NF-kappaB activation induced by lipopolysaccharide, interleukin-1, okadaic acid, phorbol 12-myristate 13-acetate, H(2)O(2), and cigarette smoke condensate as well as that induced by TNF.
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