Cart
sales@molnova.com
EN106
CAS No. 757192-67-9
EN106( ZINC3888023 )
Catalog No. M28542 CAS No. 757192-67-9
EN106 is a potent inhibitor of FEMIB and is a cysteine-reactive covalent ligand. EN106 disrupts recognition of the key reductive stress substrate of FEM1B, FNIP1.
Purity : >98% (HPLC)
COA
Datasheet
HNMR
HPLC
MSDS
Handing Instructions
| Size | Price / USD | Stock | Quantity |
| 5MG | 45 | In Stock |
|
| 10MG | 70 | In Stock |
|
| 25MG | 141 | In Stock |
|
| 50MG | 216 | In Stock |
|
| 100MG | 348 | In Stock |
|
| 200MG | Get Quote | In Stock |
|
| 500MG | Get Quote | In Stock |
|
| 1G | Get Quote | In Stock |
|
Biological Information
-
Product NameEN106
-
NoteResearch use only, not for human use.
-
Brief DescriptionEN106 is a potent inhibitor of FEMIB and is a cysteine-reactive covalent ligand. EN106 disrupts recognition of the key reductive stress substrate of FEM1B, FNIP1.
-
DescriptionEN106 is a potent inhibitor of FEMIB and is a cysteine-reactive covalent ligand. EN106 disrupts recognition of the key reductive stress substrate of FEM1B, FNIP1.
-
In Vitro——
-
In Vivo——
-
SynonymsZINC3888023
-
PathwayOthers
-
TargetOther Targets
-
Recptor——
-
Research Area——
-
Indication——
Chemical Information
-
CAS Number757192-67-9
-
Formula Weight280.71
-
Molecular FormulaC13H13ClN2O3
-
Purity>98% (HPLC)
-
SolubilityIn Vitro:?DMSO : 250 mg/mL (890.60 mM)
-
SMILESClCC(=O)N(CCC#N)c1ccc2OCCOc2c1
-
Chemical Name——
Shipping & Storage Information
-
Storage(-20℃)
-
ShippingWith Ice Pack
-
Stability≥ 2 years
Reference
1.Przegaliński E, et al. On the central antiserotonin activity of benzoctamine and opipramol. Pol J Pharmacol Pharm. 1978;30(6):781-790.
molnova catalog
related products
-
Meprednisone
Betapar(Meprednisone) is a glucocorticoid and a methylated derivative of prednisone.
-
2-Isopropyl Thioxant...
2-Isopropyl Thioxanthone has potential anti-estrogenic and anti-androgenic properties.
-
DRB18
DRB18 is a highly effective pan-class inhibitor of glucose transporter proteins (GLUT). It significantly modulates energy-related metabolism in A549 cells by inducing alterations in the abundance of metabolites associated with glucose-related pathways.
