Propagermanium
CAS No. 12758-40-6
Propagermanium( Dipropanoic acid germanium sequioxide | Ge 132 | Ge-132 | Ge132 | SK 818 | SK-818 )
Catalog No. M23442 CAS No. 12758-40-6
Propagermanium, an organometallic compound of germanium, has been used as a therapeutic agent against chronic hepatitis B in Japan.
Purity : >98% (HPLC)
COA
Datasheet
HNMR
HPLC
MSDS
Handing Instructions
| Size | Price / USD | Stock | Quantity |
| 5MG | 45 | In Stock |
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| 10MG | 68 | In Stock |
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| 25MG | 115 | In Stock |
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| 50MG | 173 | In Stock |
|
| 100MG | 258 | In Stock |
|
| 200MG | 388 | In Stock |
|
| 500MG | Get Quote | In Stock |
|
| 1G | Get Quote | In Stock |
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Biological Information
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Product NamePropagermanium
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NoteResearch use only, not for human use.
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Brief DescriptionPropagermanium, an organometallic compound of germanium, has been used as a therapeutic agent against chronic hepatitis B in Japan.
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DescriptionPropagermanium, an organometallic compound of germanium, has been used as a therapeutic agent against chronic hepatitis B in Japan.
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In Vitro——
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In Vivo——
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SynonymsDipropanoic acid germanium sequioxide | Ge 132 | Ge-132 | Ge132 | SK 818 | SK-818
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PathwayOthers
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TargetOther Targets
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Recptorothers
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Research Area——
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Indication——
Chemical Information
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CAS Number12758-40-6
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Formula Weight339.42
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Molecular FormulaC6H10Ge2O7
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Purity>98% (HPLC)
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SolubilityDMSO:Soluble
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SMILESO=[Ge](CCC(O)=O)O[Ge](CCC(O)=O)=O
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Chemical Name——
Shipping & Storage Information
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Storage(-20℃)
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ShippingWith Ice Pack
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Stability≥ 2 years
Reference
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Corticotropin-releas...
Corticotropin-releasing factor human (Human CRF; Human corticotropin-releasing factor) is an immunomodulatory neuropeptide that acts to release ACTH from the anterior pituitary and stimulates the sympathetic nervous system and adrenal medulla. Functions by reducing hyperpolarizations. Acts as a functional antagonist of inflammatory mediators.
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Piperidine-MO-1
Piperidine-MO-1 is a dopamine receptor modulator.
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Tinoridine hydrochlo...
Tinoridine (Y-3642) is a non-steroidal anti-inflammatory drug. Tinoridine (5-100 μM), produced a concentration-dependent inhibition on the simultaneous increases in lipid peroxide formation and renin release induced by 50 μM ascorbic acid in the renin granule fraction.
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