ETHYL CAFFEATE b
CAS No. 102-37-4
ETHYL CAFFEATE b( ETHYL 34-DIHYDROXYCINNAMATE )
Catalog No. M20310 CAS No. 102-37-4
In vitro ECF suppressed the differentiation of naive CD4+ T cells into Th1. Furthermore ECF intensely blocked the transcriptional expression in interferon-γ-related signaling including IFN-γ T-bet STAT1 and STAT4.
Purity : >98% (HPLC)
COA
Datasheet
HNMR
HPLC
MSDS
Handing Instructions
| Size | Price / USD | Stock | Quantity |
| 10MG | 37 | In Stock |
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| 25MG | 67 | In Stock |
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| 50MG | 112 | In Stock |
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| 100MG | 178 | In Stock |
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| 200MG | Get Quote | In Stock |
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| 500MG | Get Quote | In Stock |
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| 1G | Get Quote | In Stock |
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| 1 mL x 10 mM in DMSO | 43 | In Stock |
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| 10MG | 39 | In Stock |
|
| 25MG | 70 | In Stock |
|
| 50MG | 115 | In Stock |
|
| 100MG | 184 | In Stock |
|
| 200MG | Get Quote | In Stock |
|
| 500MG | Get Quote | In Stock |
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| 1G | Get Quote | In Stock |
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Biological Information
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Product NameETHYL CAFFEATE b
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NoteResearch use only, not for human use.
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Brief DescriptionIn vitro ECF suppressed the differentiation of naive CD4+ T cells into Th1. Furthermore ECF intensely blocked the transcriptional expression in interferon-γ-related signaling including IFN-γ T-bet STAT1 and STAT4.
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DescriptionIn vitro ECF suppressed the differentiation of naive CD4+ T cells into Th1. Furthermore ECF intensely blocked the transcriptional expression in interferon-γ-related signaling including IFN-γ T-bet STAT1 and STAT4.
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In VitroEthyl Caffeate markedly suppresses the lipopolysaccharide (LPS)-induced nitric oxide (NO) production with IC50 of 5.5?μg/ml.
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In Vivo——
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SynonymsETHYL 34-DIHYDROXYCINNAMATE
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PathwayImmunology/Inflammation
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TargetNOS
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Recptor——
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Research Area——
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Indication——
Chemical Information
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CAS Number102-37-4
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Formula Weight208.21
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Molecular FormulaC11H12O4
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Purity>98% (HPLC)
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SolubilityDMSO:41 mg/mL (196.91 mM)
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SMILESCCOC(=O)\C=C\c1ccc(O)c(O)c1
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Chemical Name——
Shipping & Storage Information
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Storage(-20℃)
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ShippingWith Ice Pack
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Stability≥ 2 years
Reference
1.Shikui X Aixue Z Zengjun G et al. Ethyl Caffeate Ameliorates Collagen-Induced Arthritis by Suppressing Th1 Immune Response[J]. Journal of Immunology Research 2017 2017:1-11.
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