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O-Phosphorylethanolamine
CAS No. 1071-23-4
O-Phosphorylethanolamine( —— )
Catalog No. M19543 CAS No. 1071-23-4
Phosphoethanolamine (PE) is a phosphomonoester metabolite of the phospholipid metabolism. PE is a precursor of phospholipid synthesis and a product of phospholipid breakdown.
Purity : >98% (HPLC)
COA
Datasheet
HNMR
HPLC
MSDS
Handing Instructions
| Size | Price / USD | Stock | Quantity |
| 100MG | Get Quote | In Stock |
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| 200MG | Get Quote | In Stock |
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| 500MG | Get Quote | In Stock |
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| 1G | 27 | In Stock |
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Biological Information
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Product NameO-Phosphorylethanolamine
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NoteResearch use only, not for human use.
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Brief DescriptionPhosphoethanolamine (PE) is a phosphomonoester metabolite of the phospholipid metabolism. PE is a precursor of phospholipid synthesis and a product of phospholipid breakdown.
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DescriptionPhosphoethanolamine (PE) is a phosphomonoester metabolite of the phospholipid metabolism. PE is a precursor of phospholipid synthesis and a product of phospholipid breakdown. Phosphomonoesters are present at much higher levels in the brain than in other organs. In developing the brain phosphomonoesters are normally elevated during the period of neuritic proliferation. This also coincides with the occurrence of normal programmed cell death and synaptic pruning in developing the brain. These findings are consistent with the role of phosphomonoesters in membrane biosynthesis. PE shows a strong structural similarity to the inhibitory neurotransmitter GABA and the GABAB receptor partial agonist 3-amino-propylphosphonic acid. PE is a phosphomonoester which is decreased in post-mortem Alzheimer's disease (AD) brain.
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In Vitro——
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In Vivo——
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Synonyms——
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PathwayOthers
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TargetOther Targets
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RecptorOthers
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Research Area——
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Indication——
Chemical Information
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CAS Number1071-23-4
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Formula Weight141.06
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Molecular FormulaC2H8NO4P
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Purity>98% (HPLC)
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SolubilityH2O:250 mg/mL (1772.30 mM; Need ultrasonic)
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SMILESNCCOP(O)(O)=O
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Chemical Name——
Shipping & Storage Information
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Storage(-20℃)
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ShippingWith Ice Pack
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Stability≥ 2 years
Reference
1.Klunk W E et al. Inactivity of phosphoethanolamine an endogenous GABA analog decreased in Alzheimer's disease at GABA binding sites[J]. Life Sciences 1995 56(26):2377.
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