Cart
sales@molnova.com
Furnidipine
CAS No. 138661-03-7
Furnidipine( —— )
Catalog No. M36174 CAS No. 138661-03-7
Furnidipine significantly reduced AoD and AF and had antiarrhythmic and cardioprotective effects at low doses in a rat model.
Purity : >98% (HPLC)
COA
Datasheet
HNMR
HPLC
MSDS
Handing Instructions
| Size | Price / USD | Stock | Quantity |
| 2MG | 164 | Get Quote |
|
| 5MG | 247 | Get Quote |
|
| 10MG | 371 | Get Quote |
|
| 25MG | 601 | Get Quote |
|
| 50MG | 854 | Get Quote |
|
| 100MG | 1134 | Get Quote |
|
| 500MG | 2277 | Get Quote |
|
| 1G | Get Quote | Get Quote |
|
Biological Information
-
Product NameFurnidipine
-
NoteResearch use only, not for human use.
-
Brief DescriptionFurnidipine significantly reduced AoD and AF and had antiarrhythmic and cardioprotective effects at low doses in a rat model.
-
DescriptionFurnidipine significantly reduced AoD and AF and had antiarrhythmic and cardioprotective effects at low doses in a rat model.
-
In Vitro——
-
In Vivo——
-
Synonyms——
-
PathwayOthers
-
TargetOther Targets
-
RecptorOthers
-
Research Area——
-
Indication——
Chemical Information
-
CAS Number138661-03-7
-
Formula Weight416.42
-
Molecular FormulaC21H24N2O7
-
Purity>98% (HPLC)
-
Solubility——
-
SMILESC(OCC1CCCO1)(=O)C=2C(C(C(OC)=O)=C(C)NC2C)C3=C(N(=O)=O)C=CC=C3
-
Chemical Name——
Shipping & Storage Information
-
Storage(-20℃)
-
ShippingWith Ice Pack
-
Stability≥ 2 years
Reference
molnova catalog
related products
-
5-Methoxysuberenone
5-Methoxysuberenone is a chemical constituent of the root bark of Toddalia asiatica (L.) Lam. (T. aculeata Pers.), Rutaceae.
-
N-Acetylglutamic aci...
N-Acetyl-L-glutamic acid (NAcGlu) is an acetylated amino acid. NAcGlu is biosynthesized from glutamic acid and acetyl-CoA by the enzyme N-acetyl glutamate synthase (NAGS).
-
5-(Hydroxymethyl)ura...
5-Hydroxymethyluracil is an oxidation damage product derived from thymine or 5-methylcytosine. It is a product of thymine dioxygenase. Additional deamination products such as 5-hydroxymethyluracil are formed when cells are under oxidative stress that is induced either by oxidizing agents or by mitochondrial dysfunction.
