9-Ethylcarbazole

CAS No. 86-28-2

9-Ethylcarbazole ( NSC 60585; NSC-60585; NSC60585; N-Ethylcarbazole )

Catalog No. M27266 CAS No. 86-28-2

9-Ethylcarbazole is an agent of biochemical.

Purity : >98% (HPLC)

COA Datasheet HNMR HPLC MSDS Handing Instructions
Size Price / USD Stock Quantity
2MG 56 Get Quote
5MG 80 Get Quote
10MG 120 Get Quote
25MG 219 Get Quote
50MG 326 Get Quote
100MG 488 Get Quote
500MG 1062 Get Quote
1G Get Quote Get Quote

Biological Information

  • Product Name
    9-Ethylcarbazole
  • Note
    Research use only, not for human use.
  • Brief Description
    9-Ethylcarbazole is an agent of biochemical.
  • Description
    9-Ethylcarbazole is an agent of biochemical.
  • Synonyms
    NSC 60585; NSC-60585; NSC60585; N-Ethylcarbazole
  • Pathway
    Others
  • Target
    Other Targets
  • Recptor
    JAK2
  • Research Area
    ——
  • Indication
    ——

Chemical Information

  • CAS Number
    86-28-2
  • Formula Weight
    195.3
  • Molecular Formula
    C14H13N
  • Purity
    >98% (HPLC)
  • Solubility
    ——
  • SMILES
    CCn1c2ccccc2c2ccccc12
  • Chemical Name
    ——

Shipping & Storage Information

  • Storage
    (-20℃)
  • Shipping
    With Ice Pack
  • Stability
    ≥ 2 years

Reference

1.Yang T, et al. N-(Pyrimidin-2-yl)-1,2,3,4-tetrahydroisoquinolin-6-amine Derivatives as Selective Janus Kinase 2 Inhibitors for the Treatment of Myeloproliferative Neoplasms [published online ahead of print, 2020 Nov 30]. J Med Chem. 2020;10.1021/acs.jmedchem.0c01488.
molnova catalog
related products
  • Unifiram

    Unifiram is a cognition-enhancer. Unifiram induces a long-lasting increase in the amplitude of field excitatory postsynaptic potentials (fEPSPs) in the rat hippocampal CA1 region (EC50= 27 nM) and increases acetylcholine (ACh) release in the rat cerebral cortex.

  • 4-Methylsalicylic ac...

    4-Methylsalicylic acid is used to inhibit the medium chain acyl-CoA synthetase.

  • VPC-18005

    VPC-18005 is a luciferase inhibitor. VPC-18005 inhibits ERG-induced transcription and interacts directly with the ERG-ETS domain thereby disrupting the ERG binding to DNA.